Neuroscience 238:34560. doi:10.1016/j.neuroscience.2013.02.005 Kajta M, Litwa E, Rzemieniec J et al (2014) Isomer-nonspecific action of dichlorodiphenyltrichloroethane on aryl hydrocarbon receptor and G-protein-coupled receptor 30 intracellular signaling in apoptotic neuronal cells. Mol Cell Endocrinol 392:9005. doi:ten.1016/j.mce.2014.05.008 Kasuya M (1974) Toxicity of phthalate esters to nervous tissue in culture. Bull Environ Contam Toxicol 12:16772. doi:10.1007/ BF01684955 Kaun-Yu L, Fu-Wei T, Chia-Jung W, Pei-Shan L (2004) Suppression by phthalates with the calcium signaling of human nicotinic acetylcholine receptors in human neuroblastoma SH-SY5Y cells. Toxicology 200:11321. doi:10.1016/j.tox.2004.03.018 Kavlock R, Barr D, Boekelheide K et al (2006) NTP-CERHR professional panel update on the reproductive and developmental toxicity of di(2-ethylhexyl) phthalate.Semaphorin-3A/SEMA3A Protein manufacturer Reprod Toxicol 22:29199 Kawano M (1980) Toxicological studies on phthalate esters. 2. Metabolism, accumulation and excretion of phthalate esters in rats (author’s transl). Nihon Eiseigaku Zasshi 35:69301 Koh JY, Choi DW (1987) Quantitative determination of glutamate mediated cortical neuronal injury in cell culture by lactate dehydrogenase efflux assay. J Neurosci Strategies 20:830.KGF/FGF-7, Human (CHO) doi:10.PMID:26644518 1016/0165-0270(87)90041-0 Kruger T, Long M, Bonefeld-J gensen EC (2008) Plastic elements impact the activation in the aryl hydrocarbon and theactivity and LDH release at micromolar concentrations. We demonstrated that the DBP mechanism of action includes ERa, ERb, PPARc, and AhR. Our study showed that AhR mediates DBP-induced apoptosis and neurotoxicity, whereas the ERs and PPARc signaling pathways are impaired by the phthalate. Nonetheless, it’s also doable that DBP activates other molecular signaling pathways. Consequently, additional studies on the mechanisms underlying the effects of DBP on the nervous system are needed.Acknowledgments This function was supported by a Grant in the Polish National Science Centre 2012/07/B/NZ4/00238. Open Access This article is distributed beneath the terms of the Inventive Commons Attribution 4.0 International License (://crea tivecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give suitable credit towards the original author(s) as well as the source, supply a hyperlink for the Inventive Commons license, and indicate if modifications were produced.
Wijerathne et al. Journal of Physiological Anthropology (2015) 34:29 DOI 10.1186/s40101-015-0065-REVIEWOpen AccessDermatoglyphics in hypertension: a reviewBuddhika TB Wijerathne1, Robert J Meier2, Thilini C Agampodi3 and Suneth B AgampodiAbstractHypertension is usually a important contributor towards the global burden of illness and mortality. A significant healthcare advancement would be a far better means to ascertain which persons are at greater danger for becoming hypertensive beforehand. To that end, there have been quite a few studies showing that specific dermatoglyphic markers are associated with hypertension. This association could possibly be explained if the danger toward establishing hypertension later on in life is somehow connected with fetal improvement of dermatoglyphics. It could be very valuable from a clinical standpoint if this conjecture may very well be substantiated since dermatoglyphic markers could then be used for screening out folks who might be at an elevated danger of becoming hypertensive. The aim of this critique was to search for and appraise offered research that pertain to the association between.