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Anxiety, frequently occurring in every day life, is really a triggering or aggravating issue of lots of diseases that seriously threaten public wellness [1]. Accumulating evidence indicates that acute pressure (AS) is deleterious to the body’s organs and systems [2, 3]. Each and every year, around 1.7 million deaths are attributed to acute injury on the kidney, certainly one of theorgans vulnerable to AS [4]. Nonetheless, to date, understanding with the etiopathogenesis and powerful preventive treatments for AS-induced renal injury stay restricted. Hence, exploring the exact mechanism of AS-induced renal injury and development of powerful preventive therapeutics is urgently required. A current study implicated oxidative stress and apoptosis in AS-induced renal injury [5]. Oxidative tension happens when2 there is an imbalance involving antioxidant depletion and excess oxides [6]. Excess oxidation products are implicated in mitochondrial harm, which triggers apoptosis [7]. Additionally, inflammation, which is mediated by oxidative anxiety, is regarded as a hallmark of kidney illness [8]. Comprehensive investigation suggests that the occurrence, improvement, and regression of renal inflammation are tightly linked to arachidonic acid (AA) metabolism [9]. Also, the anxiety hormone norepinephrine induces AA release [10]. Nonetheless, no matter whether AA metabolism is involved in a.